Search Strains

nIs190 [myo-2::GFP] integrated in or near mnC1. Approx 0.5% recombination seen between nIs190 and mnC1. Fails to complemement all markers on mnC1. Heterozygotes are WT. Segregates WT GFP+ and Egl Unc Bli; no Dpy Uncs are seen as nIs190 mnC1 homozygotes are embryonic lethal.

Egl. Dominant allele. Reference: Genetics 121(4):703-21 (1989).

Semidominant Egl.

n996 is a semi-dominant allele of egl-42. Reference: Genetics (1989) 121:703-21.

Egl. Sluggish.

Temperature sensitive Egl. Dauer defective.

Temperature sensitive Egl. Dauer defective.

Egl. [10/02: This strain was previously listed as being sel-10(n1069) or egl-41(n1069); these were found to be incorrect and the mutation is now called egl-1(n4065). H. Schwartz comm.]

Egl.

Egl.

Egl. 5HT-S, IMIP-R. Mutation causes G567E coding change. n1077 previously called egl-41.

Egl. Mate with about 50% of WT efficiency.

Egl. Grows more slowly than WT.

Dominant egg laying defective. Slightly Unc.

Egl. Semi-dominant allele. Reference: Genetics 121(4):703-21 (1989).

Semi-dominant Egl. See also WBPaper00001133.

HSN(-) Egl. Weak allele.

HSN(-) Egl.

Temperature sensitive. Wild type at 15C. Egl at 25C. HSN-. Egl.

nEx2287 [egl-6Ap::gur-3 + lin-15(+)]. Pick non-Muv to maintain. Exposure to light induces egg-laying. Reference: Bhatla N & Horvitz HR. Neuron. 2015 Feb 18;85(4):804-18. doi: 10.1016/j.neuron.2014.12.061. PMID: 25640076.

HSN-. Egl. Unc

Slightly Unc. Abnormal Q lineage. Males mate with about 50% efficiency of WT. Egl.

Unc. Dominant Egl.

Heterozygotes are Muv and Egl (n137 is semi-dominant) and segregate DpyMuv and Unc lethals (most arrest as L1-L3, the few survivors are sterile, scrawny, Unc and have a large ventral blip at hte position of the vulva). Maintain by picking non-Dpy non-Unc.

n1165 blocks cell death. Temperature sensitive Egl. Semi-dominant Egl.

Non-Egl.

Dominant Egl. Unc. Dpy (ts). Throws males.

Temperature sensitive. Sup Vul. Egl of n302.

Unc. Egl.

Unc. Egl.

n945: HSN- . Egl. Coiler.

Animals heterozygous for the duplication are WT. Animals which have lost the duplication are Unc. Animals homozygous for the duplication are viable. They are small, sickly Egl worms which don't give rise to Uncs.

ts. Dpy. Egl. sup of lin-12(d).

Bloated. Mec. Egl. Him.

Low penetrance Egl. HSN migration defective.

Egl. HSNs are migration defective and defective in serotonin expression. See also WBPaper00003422.

Sex muscles are absent. Egl.

Unc. Egl. Transformation of sex myoblasts into body wall muscle.

Egg-laying defective. Moderate to severe bloating. 30% make bags of worms. Males mate.

Dpy. Egl. Transformation of sex myoblasts into body wall muscle.

Egl. Defective in sex muscles (missing).

Absence of cell death. See WBG 10(1):31.

Egl. Unc.

Small. Egl. Slow growing.

Egl. Maintain under normal conditions. Reference: DeVore DL, et al. Cell. 1995 Nov 17;83(4):611-20.

Temperature sensitive Egl.

Egl. Maintain under normal conditions. Reference: DeVore DL, et al. Cell. 1995 Nov 17;83(4):611-20.

WT strain which segregates WT, Dpy L1 lethals, Lon males and dead eggs. Class II egl-15 mutation.