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Revertant of dominant Unc. WT.

n1279 is lethal. Animals which are alive contain the duplication. n1279 pka sup-20.

sup-17(n1258) is a recessive suppressor of lin-12(n302sd).

Temperature sensitive. Sup Vul. Egl of n302.

ts. Dpy. Egl. sup of lin-12(d).

Suppressor of lin-12(d).

Suppressor of lin-12(d).

Suppressor of lin-12(d).

sel-4(n1259) is a recessive suppressor of lin-12(n302sd).

sel-5(n1254) is a recessive suppressor of lin-12(n302sd). n1254 is cold sensitive.

Absence of cell death. Strong allele.

Wild-type revertant of n1324.

Very mildly Dpy Tc1-induced dpy-19 allele. Generates severe Dpys when Tc1 hops out of dpy-19 (doesn't excise cleanly). See also WBPaper00001672. [Some concern whether the Mut phenotype in this strain is actually mut-2 because it showed no transposition of Tc3 or other Tc's besides Tc1. 1/95.]

5% XO self-progeny.

Unc. Egl. Transformation of sex myoblasts into body wall muscle.

Egg-laying defective. Moderate to severe bloating. 30% make bags of worms. Males mate.

Dpy. Egl. Transformation of sex myoblasts into body wall muscle.

Wild type. n1412 loss-of-function suppresses the e1500sd rubberband phenotype.

Wild type. n1415 loss-of-function suppresses the e1500sd rubberband phenotype.

Absence of cell death. See WBG 10(1):31.

Dpy. Daf-C. Maintain at 15C.

HSN defective. Egl. Altered FITC loading of amphid/phasmid.

Defective in dye filling (FITC or DiO) of amphid and phasmid neurons. Chemotaxis defective.

Vul. Unc-Very sluggish as L1, moves better as adult. Weak kinker. Head region stiff. Moves better in reverse, fairly active.

Dpy. unc-93(n1415) loss-of-function suppresses the sup-9(n1550) Rubberband phenotype.

ADF fills, other amphids are defective.

Unc. Posterior half thin and pale.

Vul. Suppressor of lin-15(n765).

Vulvaless suppressor of lin-1(e1275).

n765 is temperature-sensitive Muv. Loss-of-function of sup-10(e2127) suppresses the sup-9(n1550) Rubberband mutation.

Temperature sensitive. About 50% HSN missing. Mab confirmed by Hillel Schwartz 12/5/00. Rec'd new stock from Horvitz lab 12/2000. [NOTE: The genotype of this strain was incorrectly described as carrying mab-5(mu114); however, the actual allele is mab-5(mu14).]

Vulvaless. Strong loss of function allele.

About 11% of the animals should be Vulvaless. n1779 suppresses clr-1.

At 20C about 50% of the hermaphrodites are Egl. Males have an abnormal tail and do not mate.

Unc. Muv (sd, gf).

Dead cells persist. Maternal rescue of embryonic.

Suppressor of clr-1(e1745) at 25C.

Vulvaless. Temperature sensitive. Suppressor of n765.

Vulvaless. Strong loss of function allele.

Heterozygotes are WT and segregate WT, MuvUnc (n1046 e66) and n1531 homozygotes whose phenotypes range from a few dead larva to sterile adults. The normal larval lethality of n1531/n1531 is suppressed by the maternal contribution of n1046. n1046 is semi-dominant.

Heterozygotes are mostly non-Muv (about 20% will be Muv because n1700 is semi-dominant) and non-Unc. Hets segregate Twitchers which die at various larval stages or as Vul adults, because of the maternal effect rescue of n1531 by n1700. The Twitchers will not give viable progeny. Hets also segregate DpyMuvs. The strain segregates males which will mate, though not very well.

Egg laying defective. Makes bags of worms. Backward Unc. Throws males.

Dpy (ts). Muv (sd, gf).

Dominant maternal effect. Survival of cells which normally undergo programmed cell death.

Lethal suppressor of lin-15(n765).

Unc. n2018 is cold sensitive. Most animals at 15C are Vul/Let. AT 25C, 25% of the animals are non-Vul.

n1813: dead cells persist; maternal rescue of embryonic.

Cell corpses persist. Unc. n1892 is the strongest allele of ced-7.

ced-6(n1813): dead cells persist, maternal rescue of embryonic. Unc. Long.

Engulfment of cell corpses defective. Zygotic recessive.