Search Strains

Osm. FITC filling defective.

Osmotic avoidance defective. FITC fills most amphid cells.

Semidominant osmotic avoidance defective. FITC fills normally.

Osmotic avoidance defective. FITC fills normally. Previously called osm-13.

ADF fills, other amphids are defective.

Temperature sensitive. About 50% HSN missing. Mab confirmed by Hillel Schwartz 12/5/00. Rec'd new stock from Horvitz lab 12/2000. [NOTE: The genotype of this strain was incorrectly described as carrying mab-5(mu114); however, the actual allele is mab-5(mu14).]

Adult Blistered, especially in the head. Starved translucent appearance at 20c; inviable at 25C.

Vulvaless. Strong loss of function allele.

Dead cells persist. Maternal rescue of embryonic.

Vulvaless. Strong loss of function allele.

n1813: dead cells persist; maternal rescue of embryonic.

ced-6(n1813): dead cells persist, maternal rescue of embryonic. Unc. Long.

Vul. Strong loss of function allele.

Strong allele of ced-9. Reference: (1999) Genetics 153(4):1655-71.

Roller. Long. Unc. XO Lethal. sc8 previously called rol-4(sc8).

Non-Lethal allele of let-60. Clear. Suppresses n765. Maintain at 15C.

Weak defects in protection and killing.

Egl - Bag, Con. Strong allele of sem-4.

Unc. Weak allele of unc-76.

Con, Shk(ts). Weak allele of unc-25.

Gain-of-function allele. Spontaneous shrinker, jerky, Unc. G925A, V309M in the pore lining domain. References: Jospin M, et al. PLoS Biol. 2009 Dec;7(12):e1000265. Stawicki TM, et al. Curr Biol. 2011 May 24;21(10):883-8.

Wide, meandering excretory canals, with some septate fluid-filled cysts. Canal enlargement visible from L1 through adult. Defect visible only by Nomarski microscopy. n2669 was originally listed as an allele of exc-5. Later repeated complementation tests showed n2669 to be an allele of a novel locus positioned close to exc-5.

Heterozygotes are WT and segregate WT, steriles with a vulval defect, and scrawny Dpys. n2679 is a suppressor of let-60(n1046) Muv, and is recessive sterile with vulval defects. n267 is an intermediate strength allele. See also WBPaper00002150.

n1760: strong allele of lin-39, vulvaless (n300-like). n945: HSN-. Egl. Coiler.

n2812 is a strong loss-of-function allele and a maternal effect lethal.

Weak allele of sqv-7. mid-L4 vulva abnormal. Somewhat sterile.

Temperature sensitive - maintain at 15C. Seam cells divide in L4-to-adult molt. Animals undergo extra molt and explode at vulva. Males have leptoderan-like tail. Animals explode or are sterile at 25C.

nIs31 [mec-7::ced-4a + rol-6(su1006)]. Rollers.

n1761 cuases a partially penetrant "rod-like" larval lethal phenotype (70% at 20C) and a partially penetrant vulvaless phenotype (30% at 20C). It is a splice site defect predicted to eliminate 62 amino acids at the C-terminus.

Medium strong allele. Suppressor of ced-9(n1950n2161) maternal effect lethality.

Medium-strong alllele; suppresses of ced-9(n1950 n2162) maternal effect lethality. Reference: (1999) Genetics 153(4):1655-71.

Suppressor of ced-9(n1950n2161) maternal effect lethality. Strong allele of ced-3.

n2454 is a strong allele of ced-3.

nEx648 [hsp::unc-30 (PD 49-78 and PD 49.3) + rol-6(su1006)]. Pick Rollers to maintain.

Weak allele of mek-2. 95% of the animals are WT. See also WBPaper00002150.

Suppressor of let-60(n1046). Strongest allele. Homozygous viable.

Suppressor of n1046. Weak allele of ksr-1.

Suppressor of n1046. Strong allele.

n2449 is a weak allele of ced-3.

n2424 is a weak allele of ced-3.

n3082 is a semidominant suppressor of egl-1(n1084sd) Egl- phenotype. Recessive Ced- phenotype - average of 11 extra cells in anterior pharynx. n3082 is a loss of function allele.

n1812: dead cells persist, maternal rescue of embryonic.

Not hermaphroditic. Sexes separate. Growth slow. MT received from Rothamsted Experimental Station, Harpenden, Hertfordshire, England. See also WBPaper0000567.

Kinker Unc, neuronal lineage defects including lineage reiterations and abnormal corpse pattern in ventral cord. Allele causes W113 opal.

nIs50 [mec-7::ced-3a + rol-6(su1006)]. Rollers.

Heterozygotes are WT and segregate WT, Sterile Dpys, and Mel Uncs. cup-5(n3194) is a Q139 ochre allele with a maternal effect lethal phenotype including accumulation of refractile bodies resembling apoptotic cells in some regards. cup-5 homozygotes are also defective in coelomocyte uptake.

Synthetic Muv. n433 is lin-15 Class A allele.

bwIs2 [flp-1::GFP + rol-6(su1006)]. Segregates >90% Rollers and 100% GFP+. Expresses GFP in the AVK neurons. Insertion site not mapped.

bwIs4 [fax-1::GFP + rol-6(su1006)]. Rollers. GFP reporter with expression in various neurons and DTC.

bwIs6 [nhr-67::GFP + rol-6(su1006)]. Rollers.