Laboratory Information

NameNF View on WormBase
Allele designationtk
HeadKiyoji Nishiwaki
InstitutionKwansei Gakuin University, Sanda, Japan
Address Kwansei-Gakuin University
School of Science and Technology
2-1 Gakuen, Dept. of Bioscience
Sanda 669-1337
Japan
Website http://sci-tech.ksc.kwansei.ac.jp/~nishiwaki/
Gene classes cogc  phal  saf 

Strains contributed by this laboratory

Strain Genotype Species Description
NF1226 mig-22(tk69) III/hT2 [bli-4(e937) let-?(q782) qIs48] (I;III). C. elegans mig-22(tk69): DTC migration defect and maternal effect embryonic lethal. Heterozygotes are WT with pharyngeal GFP signal. Homozygous hT2[bli-4 let-? qIs48] are inviable.
NF1684 cogc-3(k181) I. C. elegans Distal tip cell migration defective. Growth delay. Protruding vulva.
NF196 sqv-5(k172) I. C. elegans DTC migration defect.
NF198 mig-17(k174) V. C. elegans Distal tip cell migration defective.
NF199 sqv-5(k175) I. C. elegans DTC migration defect.
NF299 cogc-1(k179) I. C. elegans Distal tip cell migration defective. Growth delay. Protruding vulva.
NF317 mig-23(k180) X. C. elegans Distal tip cell migration defective. Tc1 insertion mutant. Spontaneous mutant with ventral white patch phenotype.
NF591 mig-22(tk24) III. C. elegans DTC migration defect.
NF67 mig-18(k140) III. C. elegans Distal tip cell migration defective.
NF68 mig-22(k141) III. C. elegans DTC migration defect.
NF69 mig-19(k142) II. C. elegans Distal tip cell, HSN migration defective.
NF773 fbl-1(k201) IV. C. elegans Isolated as a dominant suppressor of DTC migration defects of mig-17(k174). The fbl-1(k201) single mutant has weak DTC migration defects.
NF774 fbl-1(k206) IV. C. elegans Isolated as a dominant suppressor of DTC migration defects of mig-17(k174). The fbl-1(k206) single mutant has weak DTC migration defects.
NF78 mig-20(k148) X. C. elegans Distal tip cell, HSN, left cc mother cell, QR(d) migration defective.
NF8 mig-17(k113) V. C. elegans Distal tip cell migration defective.
NF963 fbl-1(tk45) IV/nT1 [qIs51] (IV;V). C. elegans Heterozygotes are WT and GFP+. nT1[qIs51] is probably homozygous lethal. qIs51 is an insertion of ccEx9747 with markers: myo-2::GFP expressed in the pharynx throughout development, pes-10::GFP expressed in the embryo, and a gut promoter F22B7.9::GFP expressed in the intestine. fbl-1(tk45) homozygotes are Dpy, Sterile and are Distal Tip migration defective.

Alleles contributed by this laboratory

Allele Type DNA Change Protein Change
tk69 Allele deletion
tk24 Allele substitution splice_site
tk45 Allele