More Fields
Strain Species Genotype
CB1196 C. elegans unc-26(e1196) IV. Show Description
Unc-severe kinker. Small. Scrawny. Flaccid. Little movement. Slow pharyngeal pumping.
CB205 C. elegans unc-26(e205) IV. Show Description
Growth slow. Unc and Small. Revertible. M-MATING-NO SUCCESS.
DR2 C. elegans unc-26(m2) IV. Show Description
Kinky. Coiler.
DR97 C. elegans unc-26(e345) IV. Show Description
EG3027 C. elegans unc-26(s1710) IV. Show Description
Severe kinker. Small and scrawny with flaccid little movement. Slow pharyngeal pumping.
BC11580 C. elegans dpy-5(e907) I; sEx11580. Show Description
sEx11580 [rCes JC8.10b::GFP + pCeh361]. Maintain by picking WT. WT animals are GFP+. Strain construction supported by Genome British Columbia and Genome Canada. Please acknowledge McKay et al, Cold Spring Harbor Symposia on Quantitative Biology 68: 159-169 2004 (WBPaper00006525).
CB4391 C. elegans unc-31(e928) unc-26(e2340) IV. Show Description
Unc. Eat.
DR107 C. elegans unc-26(e205) dpy-4(e1166) IV. Show Description
MT2405 C. elegans ced-3(n717) unc-26(e205) IV. Show Description
Unc. Abnormal cell death. Cells that normally die survive.
CZ4280 C. elegans eps-8(jc36)/unc-26(e205) dpy-4(e1166) IV. Show Description
Heterozygotes segregate wild-type heterozygotes, Emb, and Unc Dpy. Maintain by picking wild-type.
GS460 C. elegans evl-15(ar126) dpy-20(e1282)/unc-26(e205) IV. Show Description
Heterozygotes are WT and segregate WT, Uncs and DpySteriles with an everted vulva. Recombines. Do not distribute this strain; other labs should request it from the CGC. This strain cannot be distributed to commercial organizations. This strain cannot be used for any commercial purpose or for work on human subjects.
MT3414 C. elegans dpy-20(e1282) unc-31(e169) unc-26(e205) IV. Show Description
Dpy Unc.
BJH728 C. elegans unc-26(pek288[R216Q]) IV. Show Description
unc-26(pek288) is a CRISPR-engineered R216Q substitution in unc-26/synaptojanin 1 associated with early-onset Parkinsonism (EOP) (Quadri et al., 2013; Krebs et al., 2013). This allele shows abnormal focal accumulation of ATG-9 in presynaptic nerve terminals, defects in activity-induced synaptic autophagy, and defects in sustained neurotransmission and locomotory behaviors in aging animals. Reference: Yang S, et al. Neuron. 2022 Mar 2;110(5):824-840.e10. PMID: 35065714