Heterozygotes are Unc and segregate Uncs, dead eggs, and Pvul slow growing tm2610 homozygotes. Pick Unc to maintain and check for correct segregation of progeny. Reference: Bhalla N, et al. PLoS Genet. 2008 4(10) e1000235.

ieSi1 [htp-3p::GFP::him-8 + unc-119(+)] II. ltIs37 [pie-1p::mCherry::his-58 + unc-119(+)] IV. ieSi1 is prone to silencing; GFP might not be visible at lower magnifications. [NOTE: the ltIs37 [pie-1p::mCherry::his-58 + unc-119(+)] IV transgene was previously annotated as itIs37 in this strain. The correct name of the transgene is ltIs37 and not itIs37.] Reference: Wynne DJ, et al. J Cell Biol. 2012 Jan 9;196(1):47-64.

mIs11 [myo-2p::GFP + pes-10p::GFP + F22B7.9::GFP]. Heterozygotes are wild-type with dim GFP signal in the pharynx. mIs11 homozygotes are wild-type with bright GFP in the pharynx. ieDf2 homozygotes (non-GFP) develop normally but produce 97.5% inviable embryos and a high frequency of males among the surviving self-progeny. Pick WT with dim GFP+ in pharynx to maintain. mIs11 homozygotes will quickly overtake the population if not selected against. GFP expression in 4-cell embryos, pharyngeal muscle and gut. ieDf2 is a deficiency of zim-1, zim-2, zim-3, and him-8 generated by MosDel, resulting in single-copy insertion of a copy of the C. briggsae unc-119 gene on Chromosome IV. The deletion spans the sequences from the beginning of the zim-1 coding sequence through the ttTi22866 Mos1 insertion site.

Multi-vulva (Muv).

Unc. DNAse undetectable. Gut DNA fluoresence abnormal. M-MATING-NO SUCCESS.

Mild Twitcher Unc.

Dauer defective. Notched head. Unlinked double. M-MATING-NO SUCCESS.

Unc. Chemotaxis abnormal. Short. Variable Dpyish. M-MATING++ 1-10%WT.

Chemotaxis abnormal. Rounded head.

Chemotaxis defective: Cl-. Dauer recovery slow. Movement almost wild-type. Recessive. M-MATING+++ 10-30%WT.

Unc, coiler. Lannate sensitive.

Segregates males (5%).

3% XO self progeny.

Dpy. M-MATING+++ 10-30%WT. Weakly semi-dominant.

Unc.

Twitcher Unc.

Unc-almost paralyzed. Growth slow. Not revertible DES.

Unc-severe kinker. Small. Scrawny. Flaccid. Little movement. Slow pharyngeal pumping.

Coiler Unc. Growth slow.

Dpy.

Slightly slow Unc. Movement almost wild-type. Body muscle abnormal. M-MATING++ 1-10%WT.

Blebs on tail of adult hermaphrodite. Tail irregular, tail cuticle sometimes separated, tail spike sometimes truncated. Some animals have enlarged excretory canals.

Throws 11% males.

Paralyzed Unc. Small.

Paralyzed Unc.

Temperature sensitive. Lineage abnormal. Multi-vulva. M-MATING+POOR <1%WT.

Dpy. Temperature sensitive. Lethal cold sensitive. Male lethal. M-MATING++ 1-10%WT.

Slow but normal development, disorganized muscle structure, somewhat Egl.

Mechanosensory abnormal. Neurons/Touch abnormal. Lethargic. M-MATING++ 1-10%WT.

Dauer defective. Non-crowder. Chemotaxis normal.

Weak kinker.

Amphid defect. Chemotaxis defective (Na+). Crowds. Dauer defective. Very leaky. M-MATING+POOR <1%WT.

Vulvaless. Recessive. M-MATING+++ 10-30%WT.

15% XO self-progeny. 6% XXX self-progeny. Low brood size. 78% unhatched eggs. General non-disjunction. Recessive.

37% XO self-progeny. 6% XXX self-progeny. Recessive. M-MATING++ 1-10%WT. [Background mutation that causes rays 8 and 9 to fuse.]

Unc.

Unc.

Slow moving Unc. Recessive. M-MATING+POOR <1%WT.

Heterozygotes are WT and segregate WT, UncDaf and Lethals. Suppressed Unc in hets. Suppressed Daf in hets. sup-6 is recessive lethal.

Semi-dominant Dpy. Mapping marker standard. M-MATING++ 1-10%WT.

DpyUnc. e184 is semi-dominant.

Paralyzed Unc. Recessive. M-MATING-NO SUCCESS.

Growth slow. Unc and Small. Revertible. M-MATING-NO SUCCESS.

Levamisole resistant. Recessive. Marginally Unc-No sin wave. WT adults.

Suppressed Unc. Movement almost WT.

tra-3 suppressed. Viable hermaphrodite. See also CGC 397.

Maternal effect tra-3. Heterozygotes are WT and segregate WT and Dpy. Homozygous tra-3 animals are WT hermaphrodites which segregate abnormal sterile males or intersex.

Dominant suppressor of Unc. Movement almost WT.

Unc-cannot back. Recessive.

Abnormal cell death.