||sup-40(lb130)/+ I; unc-8(e15) IV; egl-1(n487) V.
||lb130/+ dominantly suppresses the e15 backing defect and causes recessive sterility. Three phenotypes are present in this strain: backing steriles (lb130 homozygotes), backing fertiles which are bloated (heterozygotes), and non-backing coiled Unc animals which are fertile and bloated (sup-40(+)). Maintain strain by picking fertile animals which can back. egl-1(n487) is included in the strain to facilitate identification of lb130 homozygotes, which grow slowly; their sterility contrasts with the bloating of lb130/+ heterozygotes.
||Egg-laying defective. Retains late stage eggs. Semi-dominant. Males mate successfully. Egg-laying serotinin sensitive and imipramine resistant.
||Egl. Dominant allele. Reference: Genetics 121(4):703-21 (1989).
||Egl. [10/02: This strain was previously listed as being sel-10(n1069) or egl-41(n1069); these were found to be incorrect and the mutation is now called egl-1(n4065). H. Schwartz comm.]
||Egl. Semi-dominant allele. Reference: Genetics 121(4):703-21 (1989).
||ced-3(n1165) IV; egl-1(n487) V.
||n1165 blocks cell death. Temperature sensitive Egl. Semi-dominant Egl.
||sqt-3(sc63) him-5(e1467) egl-1(n986) unc-76(e911) V.
||Dominant Egl. Unc. Dpy (ts). Throws males.
||egl-5(n945) III; him-5(e1467) egl-1(n986) V.
||n945: HSN- . Egl. Coiler.
||ced-4(n1416) III; egl-1(n986) V.
||Absence of cell death. See WBG 10(1):31.
||n3082 is a semidominant suppressor of egl-1(n1084sd) Egl- phenotype. Recessive Ced- phenotype - average of 11 extra cells in anterior pharynx. n3082 is a loss of function allele.
||egl-1(ot282) vtIs1 V.
||vtIs1 [dat-1p::GFP + rol-6(su1006)] V. Rollers. Extra cells expressing dat-1::GFP. Whole genome sequenced strain.
||F23B12.9 Homozygous. Outer Left Sequence: caagtcaagacaaggcgaca. Outer Right Sequence: cttccgacactgtaagggga. Inner Left Sequence: ttgtgcctactcctgccttt. Inner Right Sequence: tcacagtcgtttcagcgaac. Inner Primer PCR Length: 2163. Estimated Deletion Size: about 700 bp. Attribution: This strain was provided by the C. elegans Gene Knockout Project at the Oklahoma Medical Research Foundation, which was part of the International C. elegans Gene Knockout Consortium, which should be acknowledged in any publications resulting from its use. Paper_evidence WBPaper00041807