Gene Information: fbl-1

Namefbl-1 View on WormBase
Species C. elegans
Genetic positionIV:4.30 +/- 0.003 cM
Genomic positionIV: 9540806..9546252

Strains carrying this gene

Strain Genotype Description
BT14 fbl-1(hd43)/dpy-20(e1282) unc-24(e138) IV. Heterozygotes are WT and segregate WT, Steriles (hd43 homozygotes) and Dpy Uncs.
JK3297 fbl-1(q750) IV/nT1 [qIs51] (IV;V). Heterozygotes are WT and GFP+ and segregate q750 homozygotes which are GFP- sterile adults, and dead eggs. Do not distribute this strain; other labs should request it from the CGC. This strain cannot be distributed to commercial organizations. This strain cannot be used for any commercial purpose or for work on human subjects.
NF773 fbl-1(k201) IV. Isolated as a dominant suppressor of DTC migration defects of mig-17(k174). The fbl-1(k201) single mutant has weak DTC migration defects.
NF774 fbl-1(k206) IV. Isolated as a dominant suppressor of DTC migration defects of mig-17(k174). The fbl-1(k206) single mutant has weak DTC migration defects.
NF963 fbl-1(tk45) IV/nT1 [qIs51] (IV;V). Heterozygotes are WT and GFP+. nT1[qIs51] is probably homozygous lethal. qIs51 is an insertion of ccEx9747 with markers: myo-2::GFP expressed in the pharynx throughout development, pes-10::GFP expressed in the embryo, and a gut promoter F22B7.9::GFP expressed in the intestine. fbl-1(tk45) homozygotes are Dpy, Sterile and are Distal Tip migration defective.
NK2579 fbl-1(qy62[mNG+loxP::fbl-1]) IV. Superficially wild-type. CRISPR/Cas9 insertion of mNeonGreen. Insertion site verified by PCR and sequencing.
VC642 fbl-1(gk295) IV/nT1 [qIs51] (IV;V). F56H11.1a. Homozygous lethal deletion chromosome balanced by GFP-marked translocation. Heterozygotes are WT with pharyngeal GFP signal, and segregate WT GFP, arrested nT1 aneuploids, and non-GFP gk295 homozygotes (some make it to sterile adults). nT1[qIs51] homozygotes inviable. Pick WT GFP and check for correct segregation of progeny to maintain. Attribution: This strain was provided by the C. elegans Reverse Genetics Core Facility at the University of British Columbia, which is part of the international C. elegans Gene Knockout Consortium, which should be acknowledged in any publications resulting from its use. Paper_evidence WBPaper00041807